Figure 4
From: JNK activation is responsible for mucus overproduction in smoke inhalation injury
Immunohistochemistry of the MUC 5AC protein. Wild-type smoke inhalation mice showed increased MUC5AC protein expression in their airway epithelium 4 and 24 h after injury, whereas the JNK inhibitor and JNK -/- mice groups did not (MUC5AC protein staining: A-G, 100×; H- and I, 400×). A, WC; B, WS4; C, WS24; D, JKOS4; E, JKOS24; F, JIS4; G, JIS24; H, WS4 400×; I, WS24 400×.