Figure 5From: 17β-estradiol suppresses lipopolysaccharide-induced acute lung injury through PI3K/Akt/SGK1 mediated up-regulation of epithelial sodium channel (ENaC) in vivo and in vitroEffects of 17β-estradiol (E 2 ) on total protein expression levels of alveolar epithelial sodium channel (ENaC) in mouse lungs (a) and MLE-12 cells (b) after LPS treatment. Protein level was normalized to β-actin. Data are presented as means ± S.E.M (*p < 0.05 compared with the control group, #p < 0.05 compared with the LPS group, Δp < 0.05 compared with the 17β-estradiol group).Back to article page