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Correction to: Cigarette smoke exposed airway epithelial cells-derived EVs promote pro-inflammatory macrophage activation in alpha-1 antitrypsin deficiency
Respiratory Research volume 24, Article number: 266 (2023)
The Original Article was published on 06 September 2022
Correction to: Khodayari et al. Respiratory Research (2022) Sep 6;23(1):232
After the publication of this original article [1], the authors identified an error in Fig. 3B. The images of GAPDH control was mistakenly placed within the control group into the CS group. The authors confirm that these corrections do not change the result interpretation or conclusions of the article and apologise for this error
The correct version of figure is given below.
Induction of ER stress and NF-?B inflammatory pathway in AATD macrophages in response to cigarette smoke. A Comparison of the mRNA expression levels of XBP-1, CHOP, ATF 4, and BiP in normal and AATD macrophages exposed to air or cigarette. B Western blot analysis of 4 different normal and AATD macrophages incubated with control or CS exposed showing activation of NF-?B pathway in AATD macrophages after CS exposure. C Bar graphs showing the results of quantification and normalization of band intensities, *p < 0.05, **p < 0.005, ****p < 0.00005
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The online version of the original article can be found at https://0-doi-org.brum.beds.ac.uk/10.1186/s12931-022-02161-z
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Khodayari, N., Oshins, R., Mehrad, B. et al. Correction to: Cigarette smoke exposed airway epithelial cells-derived EVs promote pro-inflammatory macrophage activation in alpha-1 antitrypsin deficiency. Respir Res 24, 266 (2023). https://0-doi-org.brum.beds.ac.uk/10.1186/s12931-023-02571-7
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DOI: https://0-doi-org.brum.beds.ac.uk/10.1186/s12931-023-02571-7